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Overexpression of ribosomal proteins, particularly Rpl15, which gradually increased from the 1-cell to 8-cell stages, indicated persistent hyperactivation of ribosome biogenesis, which promoted tumorigenesis in offspring derived from oxidatively damaged IVF embryos by selectively enhancing the translation of -Catenin and TGF-1

Therefore, our findings suggest that VEGF predominantly increased the activation of p38MAPK/STAT1 via VEGFR2, thus leading to induction of apoptotic cell death in HG-treated hRECs ( Fig

The combination of dihexa and HGF essentially eliminated labeling of membrane-bound E-cadherin (10.30.5