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The pathogenesis of childhood mitochondrial-associated epilepsy is highly complex, involving a range of genetic mutations that lead to various seizure types, often resulting in drug-refractory epilepsy

Hpb -GDH was effective at suppressing inflammatory pathways in both mouse and human macrophages and granulocytes by inhibiting the 5-lipooxygenase and instead promoting the cyclooxygenase pathway leading to the synthesis of prostanoids and the downregulation of 5-LOX metabolites ( Nematode-derived cysteine protease inhibitors (cystatins) also have demonstrated anti-inflammatory functions

NLRP3 inflammasome is a member of the innate immune system which initiates DCM when its functions are dysregulated

We highlight the non-canonical roles of multifunctional metabolic enzymes in signaling and redox control, the impact of spatial and temporal metabolic heterogeneity in shaping mitochondrial function, and the post-transcriptional regulation of mitochondrial metabolism by miRNAs in CRC