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Rather than functioning as isolated antioxidant modules, these pathways form a highly interconnected redox buffering network in which compensatory capacity, metabolic flux, and iron availability collectively determine ferroptotic susceptibility

Additionally, several other investigational compounds, including quercetin, morroniside, rapamycin, and iron chelators such as alpha-lipoic acid (ALA), have demonstrated neuroprotective properties by mitigating ferroptosis (Lin et al., 2022

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