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conducted a study that revealed the accumulation of -KG within cells to be a catalyst for the promotion of oxidative stress in lymphoma cells, resulting in the generation of substantial quantities of ROS, elevated levels in ROS promote cellular iron death by instigating lipid peroxidation and TP53 activation [52]

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3c), compounds 1 - 5 were found to be slow inhibitors as they displayed little to no inhibition after 15 min, but time-dependent inhibition of TGR over 25 h (Fig