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Instead, it creates a pro-healing environment in which the body can rebuild more effectively

Under inflammatory stimulation, SENP5 depletion further increased ROS accumulation compared with that in the TNF-+si-NC group, demonstrating that SENP5 deficiency exacerbates ROS generation during pathological stress (Fig

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When FOXO4-DRI was applied to senescent Leydig cells, it caused p53 to be excluded from the cell nucleus and induced apoptosis in these cells