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Available from: Johannsen P, Ostergaard K, Christensen JE, Worm M, Rasmussen K

A more promising approach involved boosting the expression and activity of GLO I, a catalyst in MG metabolism

In recent years, the hepatotoxicity of cantharidin has also been systematically investigated

For instance, in M1 pro-inflammatory macrophages (Ms), there is a metabolic break at isocitrate dehydrogenase in the TCA cycle due to suppression of its mRNA expression, leading to the accumulation of citrate and itaconate, which drive lipid synthesis and stabilization of anti-inflammatory transcription factors, such as nuclear factor-erythroid factor 2-related factor 2 (NRF2) and activating transcription factor 3 (15)