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In the LPS-induced acutelung injury mouse model, treatment of GHK-Cu also significantly suppressed TNF- and IL-6 expression with a significant decrease of both NF-B p65 and p38 MAPK signaling, suggesting that GHK-Cu is useful for the development of novel anti-inflammatory therapies [22]

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Studies using fetal lung fibroblasts found GHK induces dose-response gene expression changes in 329 genes associated with extracellular matrix composition

Our full overview of GHK-Cu and its clinical applications at Perfect B covers its mechanism, dosing, and the patient profiles where it consistently produces results