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Linked thioredoxin-glutathione systems in platyhelminth parasites: alternative pathways for glutathione reduction and deglutathionylation

Because mtDNA depletion or OXPHOS impairment profoundly alters redox state, membrane potential, ATP production, and metabolite distribution between mitochondria and cytosol [80], mitochondrial dysfunction can act as a source of epigenetic perturbation interventions that selectively inhibit OXPHOS complexes or mitochondrial carrier transporters can therefore help define the molecular identity of these mitochondria-derived epigenetic cues

2024;9(1):e0067223

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