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deficient energy intake may manifest as hyperkalemia due to catabolism as K + is released due to tissue breakdown [31, 36]

Furthermore, investigations have proposed that the presence of the ApoE4 allele in MS patients may contribute to disease progression, leading to heightened brain damage, exacerbated cognitive dysfunction, and augmented disease severity (Parmenter et al., 2006

Collectively, GLPs demonstrate robust therapeutic potential against a spectrum of cardiac disordersincluding diabetic cardiomyopathyby engaging multiple complementary mechanisms: activation of the PPAR/PGC-1 axis to improve mitochondrial biogenesis and energy metabolism

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