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Similar abnormalities are seen in erythrocyte sedimentation rates (ESR) [372, 373], as well as in other conditions such as delayed cerebral ischemia following subarachnoid hemorrhage [374]

Here we show that HGF adopts an enhanced form with dynamically increased receptor-binding affinity and nitration-dysfunction resistance through interaction with lipoic acid trisulfide (LASSS) under physiological conditions