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GSH depletion can trigger ferroptosis, which has been exploited in cancer treatments [17], and supplementation with GSH is utilized by glutathione peroxidase 4 (GPX4) to prevent lipid peroxidation, in turn inhibiting ferroptosis

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The presence of the two signature features of an aggregation prone protein, namely a hydrophobic region and a sequence with a strong propensity to adopt a beta pleated sheet structure make -synuclein an ideal candidate for fibrillation