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Description

Cardioprotective potential of transcription factor PRRX1 silencing against myocardial ischemia/reperfusion injury by regulating excessive mitophagy and ferroptosis through FKBP5-p38 MAPK axis

A clinico-therapeutic analysis

10.1016/s2589-7500(20)30107-2 125

We also determined whether DMB could inhibit TNBS-induced proinflammatory cytokines, endothelial nitric oxide synthase (eNOS), iNOS, TNF-, COX-1, interleukin-6 (IL-6), and COX-2 gene expression