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In PD models, downregulated Bcl-xL expression impairs the resistance of dopaminergic neurons to mitochondrial damage, whereas exogenous upregulation of Bcl-xL markedly alleviates neuronal apoptosis [38, 39]

However, several rounds of whole-genome duplication in teleosts species (Glasauer and Neuhauss, 2014) have resulted in the presence of several paralogous genes, some of which may have resulted in divergent functions