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Ongoing development of preclinical optic neuropathy models should prioritize those that can better mimic the natural progression of human disease and demonstrate the effect of aging on the survival and integration of donor cells in the retina

It has been demonstrated that ROS activates JNKs (c-Jun amino-terminal kinases) and p38 pathways

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[41] also found that combined FEP and unmedicated schizophrenia patients had elevated Glx levels in the hippocampus ( g = 0.47) and dorsolateral prefrontal cortex ( g = 0.25), but not in the dorsal anterior cingulate cortex (dACC