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Altogether these data support the notion that these therapeutic bounties are driven by the agonistic stimulation of myocardial and extra-myocardial CD36 and/or GHSR1a receptors by activating survival pathways, downregulating fibrogenic cytokines, and optimizing energetic homeostasis shunts that cooperate in cellular survival (Hosoda, 2022

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doi: 10.1016/j.phymed.2013.02.006 352 GunesHGulenDMutluRGumusATasTTopkayaAE

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To visualize the nucleus, it was stained with DAPI solution added to a solution that extends the emission of the fluorophores (ProLong Glass Antifade Mountant)

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[DOI] [PubMed] [Google Scholar] 305.Watts RJ, Sarasa J, Loge FJ, Teel AL

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