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In the context of lung cancer, this dysregulation is marked by pronounced abnormalities in DNA methylation, typically characterized by a coexistence of genome-wide hypomethylation alongside specific hypermethylation of tumor suppressor gene promoters, including Death-associated protein kinase (DAPK), Ras association domain family member 1A (RASSF1A), and Retinoic acid receptor (RAR), among others (44, 45)

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Il17a forward: 5-TGA CCC CTA AGA AAC CC CCA-3 and reverse, 5-TCA TTG TGG AGG GCA GAC AA-3