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Interestingly, the same research group recently identified an anti-inflammatory role for p62, a modulator of the KEAP1NRF2 axis, in the same DKO* model [9], suggesting a broader oxidative-stress circuitry governing psoriatic inflammation

A recent study found that SAT1 (the gene encodes SSAT) is a transcriptional target of p53 and participates in p53-mediated ferroptosis (Ou et al., 2016)

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VNella flips the script by converting that lactic acid into beneficial short-chain fatty acids (SCFAs) that power your mitochondria and provide anti-inflammatory effects