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Results TNBS Depletes GSH and Generates ROS In Vitro Although, the induction of colitis by TNBS administration is a well defined model of IBD commonly used to uncover pathophysiological factors, the interaction between GSH and TNBS, remains unknown

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Therefore, the development of drugs that target common points of these crosstalk pathways is a novel approach to antagonize cisplatin-induced ototoxicity without affecting antineoplastic efficacy

Taken together, the development of chronic inflammatory processes may represent a pivotal component in the airway remodeling observed in individuals affected by both obesity and asthma