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Gut dysbiosis-induced overproduction of LPS and peptidoglycans potently activates innate immune receptors, driving systemic inflammation that can breach the BBB and exacerbate neural damage (Kesika et al., 2021)

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Patient subgroups, characterized by distinct symptom profiles and comorbidities, exhibited increased responses to specific treatments, e.g., a POTS-dominant cluster benefiting from autonomic modulators and a cognitive-dysfunction cluster from CNS stimulants