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These interactions highlight the ability of polyphenols to simultaneously address multiple pathological mechanisms in AD, including oxidative stress, neuroinflammation, and neurodegeneration, underscoring their potential as multitarget therapeutic agents for AD

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Pharmacokinetics and Metabolic Stability in Animal Models After sc dosing in rats (0.11.0 mg/kg) and cynomolgus monkeys (0.030.3 mg/kg), peak plasma concentrations were reached within 812 h and modulated food preference, reduced food intake, and caused weight loss without decreasing energy expenditure [41,42,43]