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Apoptosis can be triggered by endoplasmic reticulum (ER) stress in which glucose-regulated protein 78 (GRP78), CCAAT-enhancer-binding protein homologous protein (CHOP) and activating transcription factor 4 (ATF4) are induced by unfold protein response (UPR) to ameliorate ER stress by stimulation of apoptosis (Di Conza and Ho, 2020)

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Its extracts may regulate cytokines such as IL-6, IL-5, IL-13, IL-17, and IL-1 by influencing the NF-B pathway (28)

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