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Although initial studies have begun to delineate its regulatory landscape and interactions with PTM, systematic exploration of this modification remains in its early stages

In a murine model of acute myocardial infarction, myeloperoxidase was found to be a major enzymatic source of acrolein, as demonstrated by a dramatic increase of acrolein in ischemic cardiac tissue obtained from wild-type mice as compared to myeloperoxi-dase-deficient (MPO null) mice [48]

Professionals need to work together to ensure every treatment offered is safe, trustworthy, and effective