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doi: 10.1007/s12016-015-8473-z 165 ClanchyFILBorgheseFBystromJBalogAPennHHullDNet al

The majority of plasmatic GSH originates from the liver, and for this reason an impairment in hepatic GSH synthesis has a systemic impact on redox balance and oxidative stress (1, 6, 7, 11)

When you have enough antioxidants around during oxidation (i.e., a high level of Glutathione), those electrons can be supplied back quickly enough and in sufficient quantity that no significant damage will result

Results: 197 patients were included in the cohort: 39% alcohol-related cirrhosis, median MELD-Na 14 (range 6-40), 34% Childs Classification C, 65% ascites, 46% encephalopathy