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The PI3K inhibitor treatment inhibits GBM invasion and angiogenesis, but it will also activate autophagy

IGF-1 LR3 is a direct IGF-1 receptor agonist it bypasses the pituitary and liver entirely and acts directly on IGF-1 receptors in muscle, fat, and connective tissue

Also, NG2 glia and neurons are not as innocent victims as they seem, because neurons participate in myelin/oligodendrocyte damage and NG2 glia lose their ability to replace dying oligodendrocytes