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31 Further, enrichment analyses indicated significant differential expression of 24 gene ontology pathways (i.e., biological processes, cellular components, or molecular functions) in epithelial cells isolated from individuals with and without AUD, including those implicated in protein targeting to the membrane and viral gene expression

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Targeted depletion of T cells and neutrophils attenuates this maladaptive immune activation, mitigates tubular injury and atrophy, and limits fibrosis progression, underscoring a central role for macrophage-driven T cell responses in perpetuating chronic inflammation [56]