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Phase 1 is believed to correspond to the high level of activity in the primary afferent as by C-fiber activation due to peripheral stimulation, 9 whereas phase 2 seems to result from the continuous low level of small afferent input, together with a facilitated state of spinal processing

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7ac, Supplementary Tables 7 and 8) and led to greater relative engagement of this peptide with TMs 5, 6 and 7, relative to rAmy, which interacted more with ECL2 (Supplementary Fig