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Although still in its early stages, the therapeutic application of biased ligands will advance through structural analysis, enabling precise modulation of receptor signaling

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The thyroid dyshormonogenesis in nrf2a 5 homozygous mutant embryos is a cell-autonomous phenotype Although the initial characterization of the nrf2a5 mutant phenotype indicates a role of nrf2a during thyroid functional maturation, we could not rule out the possibility that this phenotype is secondary to another defect affecting the whole zebrafish embryos

That is anecdotal