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Inhibiting CHAC1 could disrupt these pathways, leading to unintended consequences such as impaired cellular stress responses and the potential exacerbation of oxidative damage in non-target tissues

Serious but rare risks include pancreatitis and gallbladder disease, which require urgent medical attention

Conversely, FMNL2 interacts with Cdc42 GTP via multiple hydrophobic and polar contacts formed between all five armadillo repeats of FMNL2 and the two switch regions of Cdc42

Such necrosis typically arises from coronary artery obstruction, often due to an atherosclerotic clot or arterial spasm (Slavich and Patel, 2016)