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From the table, it can be deduced that GSH almost completely disappeared by SI2 in both LGSH and HGSH animals, with disappearance equaling 97.5 and 98.0% on d5, and 97.6 and 98.2% on d14, respectively

This TNF- pressure, in turn, drives endothelial RIPK1/RIPK3/MLKL activation and bloodspinal-cord-barrier breakdown, linking ferroptotic inflammation to necroptotic execution across compartments