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Targeting oncogenic MAGEA6 sensitizes triple negative breast cancer to doxorubicin through its autophagy and ferroptosis by stabling AMPK1

mtROS-mediated Akt/AMPK/mTOR pathway was involved in copper-induced autophagy and it attenuates copper-induced apoptosis in RAW264.7 mouse monocytes

Comparative analysis reveals this duality arises from four mechanistic distinctions ( Figure 5 ): (1) Temporal kineticsexogenous donors produce high peak concentrations with short durations possibly preferentially targeting MDSC redox sensitivity, whereas endogenous enzymes continuously produce H 2 S enabling chronic immune reprogramming

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