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Studies have shown that in breast cancer, ferroptosis can also modulate the polarization of macrophages within the tumor microenvironment, suppressing the function of M2 macrophages and consequently reducing cancer cell migration and invasion (35)

melanogaster , AGEs accumulate with age and can be experimentally modulated, supporting their use as an informative glycoxidative-stress endpoint in this model [26]

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Cytokine measurements showed that the serum levels of IL-6, IL-1 and TNF- were all elevated in CT26 tumor-bearing mice compared with normal control mice (Fig