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Genetic inactivation of components in the necrosis pathway (calpains and cathepsins) can delay worm death, confirming a role for systemic necrosis in starvation-induced organismal death

The significant accumulation of ROS has the potential to inhibit the proliferation of tumor cells and induce apoptosis or necrosis, which can be attributed to the activation of various pathways, including endoplasmic reticulum stress, mitochondrial dysfunction, P53-mediated apoptosis, ferroptosis, and other mechanisms