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The molecular mass of TTR was independently confirmed by time-of-flight liquid chromatography-mass spectrometry (LC/MS TOF 6230B, Agilent, Santa Clara, CA, USA)

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We assumed that cells were more rely on GSR-mediated recycling of oxidized glutathione to maintain GSH homeostasis in the condition that function deficiency in de novo synthesis mediated by sorafenib, which provided a feasible approach for overcoming the drug resistance