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Mice with DLX1 knockout exhibit age-dependent reductions in interneurons, weakened GABAergic synaptic currents, and eventually develop epilepsy and social abnormalities (111)

The Schwartz patent references the use of the measured levels of constituents of the pathway as indicators of the need to administer the constituents of the SAM pathway, and references cysteine and glutathione as an indicator of dysfunction of the SAM pathway

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10.1016/j.phymed.2024.155518 124 WangY.BaiM.WangX.PengZ.CaiC.XIJ.et al (2024b)