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Description

This enhanced release is attributed to the ability of MA to cleave internal -D-(1,6) and -D-(1,4) glycosidic bonds

This delay contributes to what is widely recognized as clinical inertiathe failure to initiate or intensify therapy when indicated

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Therefore, incorporating EB would likely enhance, rather than reduce, the number of significant hits we expect to detect in high contrast, small sample scenarios more so than in large sample studies