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Highlights The primary mechanism of hepatocyte death induced by Crizotinib is through ferroptosis, and the utilization of ferroptosis inhibitors effectively reverses Crizotinib-induced hepatotoxicity

This coating is also responsible for the high storage stability of GSH and its good protection against oxidation

370 In HCC, CDKN2A promotes tumor cell proliferation and migration capabilities as well as attenuating cuproptosis

Following the success of SLE, clinical research has extended the use of anti-CD19 CAR T-cell therapy to other autoimmune conditions, such as systemic sclerosis (101103), refractory immune-mediated necrotizing myopathy (103), refractory anti-synthetase syndrome (104, 105), and myasthenia gravis (96)