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have conducted a review of the literature and revealed that preliminary findings from studies looking at similar GHRH analogs and GHS-R agonists support this concept

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Animal toxicology studies have not revealed significant adverse effects at therapeutic dose ranges

The primary underlying pathomechanism is most likely a decreased hepatic GHR expression related to insulin deficiency, resulting in GH-resistance and impaired hepatic IGF-1 synthesis, which stimulates pituitary GH-secretion by a feedback mechanism [188, 189]