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In plants, stress conditions can deplete free GSH which is partially due to its utilization in GSH-dependent detoxification of RCS as well as of methylglyoxal and glyoxal

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In vitro investigations have demonstrated that the aberrant activation of the MAPK signaling pathway in BRAF V600E-mutant TC can upregulate the production of DNMT1, thereby inducing hypermethylation of the TSHR promoter and leading to its transcriptional suppression [34, 35]

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