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Abstract This study has been initiated to investigate whether sunitinib (SUN) alters the expression of key genes engaged in mitochondrial transport and oxidation of long chain fatty acids (LCFA), and if so, whether these alterations should be viewed as a mechanism of SUN-induced cardiotoxicity, and to explore the molecular mechanisms whereby carnitine supplementation could attenuate SUN-induced cardiotoxicity

Variants located in the coding region and splicing sites were analyzed

Hurdle or thruster: glucose metabolism of T cells in anti-tumour immunity

doi: 10.1186/s12957-022-02777-x 57 WuBChenYChenYXieXLiangHPengFet al