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These results confirmed the beneficial effects of allicin on cell death and survival and the inflammatory response

ROS-induced mtDNA damage impairs mitochondrial OXPHOS, forcing tumor cells to rely on glycolysis for ATP production ( Figure 1 2.1 Mitochondrial metabolic regulation of immune cells T cells rely on OXPHOS and fatty acid oxidation in the resting state, but switch to aerobic glycolysis and fatty acid synthesis upon activation to support proliferation ( Figure 2 )

Improving GSH levels by increasing precursor availability may be important to the metabolic control of diabetic patients since it improved glucose disposal rate and insulin sensitivity, possibly by decreasing free fatty acids and reactive oxygen species as shown in HIV-infected patients [22]

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