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It has been reported that EGLN2 (Egl-9 family hypoxia inducible factor 2) is a candidate driver of iron chelation-mediated inhibition of cell death, and the hydroxylase activity of EGLNs is iron-dependent[114, 115]

doi:10.1007/s12016-020-08791-9 Kosowski M, Smolarczyk-Kosowska J, Hachula M, et al

The respective contributions of a wider range of chromatin remodelers to cohesin loading, individually or in combination, remains to be further investigated