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262 Importantly, the mitophagic response in such cells requires both mTORC1 inhibition and loss of mitochondrial membrane potential, which activate PARKIN recruitment to mitochondria

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A prominent example is their inhibition of the PI3K/Akt pathwayGA treatment in lung cancer cells led to downregulation of PI3K/Akt alongside increased p53 levels, which in turn activated downstream executioner caspases and cell cycle inhibitors (p21Cip1/p27Kip1), culminating in apoptosis and growth arrest (151)

However, research into glutathione benefits remains scarce