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The resulting Ca + overload disturbs ion homeostasis and induces mitochondrial dysfunction, oxidative stress, and neuronal edema, ultimately culminating in cell death ( Studies have shown that RIPreC significantly reduces glutamate levels in both the ischemic core and penumbra, mainly through the enhanced efflux of glutamate into peripheral blood

Prior studies have reported SalA-mediated cardioprotection, hepatoprotection, and nephroprotection through mitigation of ROS and inflammatory burden (Youssef et al