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Jun 7 2017;18(6)doi:10.3390/ijms18061221

While the functional relevance of suboptimal riboflavin status in otherwise healthy individuals is not clear, in vitro studies have shown increased protein carbonylation, DNA strand breaks, and cellular stress and apoptosis with riboflavin deficiency associated with little to no glutathione reductase activity (211, 340)

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NAPstar6 and 7, with a high K r(NADPH/NADP+) , showed a limited response with a rapid recovery, while the TS/mC ratio of NAPstar4.3 was close to that of NAPstarC (Supplementary Fig