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Mitochondrial Outer-Membrane Protein FUNDC1 Mediates Hypoxia-Induced Mitophagy in Mammalian Cells

Because this work examines direct TAM effects on experimental endpoints only in the context of neurogenesis research in adult animals, further testing is needed to determine the suitability of alternate TAM dosing methods for subadult animals and research on other brain processes

Despite its strong potential to treat DM, NS and TQ must be examined in more inclusive clinical studies targeting underrepresented patient populations

Second, KBs have been recently shown to inhibit ASIC1a channels, but the precise mechanism and the relevance of these channels in epileptic contexts remains an open question (Zhu et al., 2019)