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Although functional loss of SRs can result in hypercholesterolemia, which leads to atherosclerosis and heart disease, it has been shown that when the SR-B2 is exposed to modified LDL, it interacts with TLR4 and TLR6, resulting in NF-B activation and contributing to the inflammatory response associated with atherosclerosis and promoting necrotic lesions of plaques (163, 164)

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Ipamorelin, on the other hand, is a selective GHRP (Growth Hormone Releasing Peptide) that acts on ghrelin receptors, amplifying GH release in a controlled, physiological pattern