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Combinations of substrates that can provide the components of ATP (ribose, phosphate, adenine) and the energy for ATP restoration have been suggested to support ATP restoration in ATP-deprived brain cells [235]

The UPR produces upstream activation signaling that triggers autophagy [35], which activates ATF4, inositol-requiring enzyme 1 (IRE1), and other molecules to mediate ADCD after pharmacological treatment [36, 37]

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Many AI models, particularly DL models, are often regarded as black boxes due to the difficulty in explaining their internal mechanisms