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It is generally believed that an increased amphetamine climbing behavior is a result of striatal dopamine receptor upregulation following dopamine antagonist haloperidol application

Most of all, these phenomena are relevant to the activity of the AMPA receptor signaling pathway, which launches a cascade of reactions contributing to persistent LTP

Experimental studies in animal models and in vitro systems suggest that DSIP can influence the release of hormones such as growth hormone, luteinizing hormone, and adrenocorticotropic hormone through both hypothalamic and pituitary-level mechanisms [8]

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