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MOTS-c has been demonstrated to disrupt the folate-methionine cycle and impede de novo purine biosynthesis, and it also regulates the expression of genes such as GLUT4, STAT3, and IL-10

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These findings suggest potential red blood cell morphology alterations in FM patients, which may indicate underlying metabolic or inflammatory changes

There is a high heterogeneity of ECs surface and cytoplasmic markers [9]