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Autoimmune reactions to oxidized LDL are critical factors contributing to atherosclerosis, and autoimmune responses to oxidation-specific epitopes may exacerbate atherosclerotic processes [182]

For example, targeting metabotropic receptors or transporters that control the release of neurotransmitters that are perturbed early in PD could prevent neurodegeneration

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Gut microbiota production of trimethyl-5-aminovaleric acid reduces fatty acid oxidation and accelerates cardiac hypertrophy [J]